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About this product
- PublisherSpringer Basel
- Date of Publication01/11/2012
- GenreLife Sciences: General
- Series TitleAgents and Actions, Supplements
- Series Part/Volume Number18
- Country of PublicationSwitzerland
- ImprintSpringer Basel
- Content Note20 black & white illustrations, biography
- Weight294 g
- Width170 mm
- Height244 mm
- Spine9 mm
- Edition StatementSoftcover reprint of the original 1st ed. 1986
- Table Of ContentsContributors.- Changes in Cartilage Proteoglycan Structure During Ageing: Origin and Effects - A Review.- The Mechanisms of Cartilage Catabolism.- The Possible Role of Synovial Factors in Cartilage Destruction.- Mononuclear Cell Factor Modulates the Concomitant Release of Proteoglycan Latent Proteinase by Human Articular Cartilage in Organ Cutlure.- Responses of Human Synovial Fibroblasts to Mononuclear Cell Factors and Retinoic Acid.- The Effects of Some Polysulphated Polysaccharides on Hyaluronate (HA) Synthesis by Human Synovial Fibroblasts.- Degradation of Cartilage Matrix by Purified Elastase and Its Control by an Endogenous Purified Specific Cartilage Elastase Inhibitor.- Variation with Ageing and Degeneration of the Serine and Cystein Proteinase Inhibitors of Human Articular Cartilage.- Neutrophil Enzymes, Antiproteases, and Oxygen Radicals: Interaction in Cartilage Proteoglycan Degradation and Effect of D-Penicillamine.- Amplification of Oxyradicals by Eicosanoid Synthesis.- The Generation of Reducing ends by Exposure of Hyaluronic Acid to Oxygen Derived Free Radicals.- An Assay for Proteoglycanase and Its Application to Cultured Articular Chondrocytes.- Fine Epitope Spicifity of Murine Monoclonal Antibodies Reacting with Rabbit Cartilage Proteoglycan.- Articular Cartilage Proteoglycans Immunoreactive with an Antibody to Skin Proteodermatan Sulfate Core Protein.- Identification and Isolation of the Major Glycoprotein Antigen Associated with Elastic Fibre Microfibrils.- Increased Prevalence of Mitral Valve Prolapse Associated with an Elevated Skin Type III/III+I Collagen Ratio in Joint Hypermobility Syndrome.- The Effect of Interleukin-l on Connective Tissue Metabolism and Its Relevance to Arthritis.
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